Erika Piedras-Renteria, PhD

Cell and Molecular Physiology

Associate Professor

Languages Spoken

English, Spanish

Biography

Biography

Primary fields of interest:
Neuronal excitability, voltage-gated calcium channel function and dysfunction, calcium channel biophysics

Interests
Pottery, Art History

Board Certification

American Nurses Credentialing Center
Anesthesiology
American Board of

Research

  • Calcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice Perissinotti, P. P.; Ethington, E. A.; Almazan, E.; Martinez-Hernandez, E.; Kalil, J.; Koob, M. D.; Piedras-Renteria, E. S., Frontiers in cellular neuroscience
  • Down-regulation of endogenous KLHL1 decreases voltage-gated calcium current density Perissinotti, P. P.; Ethington, E. G.; Cribbs, L.; Koob, M. D.; Martin, J.; Piedras-Renteria, E. S., Cell calcium
  • Elimination of the actin-binding domain in kelch-like 1 protein induces T-type calcium channel modulation only in the presence of action potential waveforms Aromolaran, K. A.; Benzow, K. A.; Cribbs, L. L.; Koob, M. D.; Piedras-Renteria, E. S., Journal of signal transduction
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  • Forced-exercise delays neuropathic pain in experimental diabetes: effects on voltage-activated calcium channels. Shankarappa, S. A.; Piedras-Renteria, E. S.; Stubbs, E. B., Jr, Journal of neurochemistry
  • T-type current modulation by the actin-binding protein Kelch-like 1. Aromolaran, K. A.; Benzow, K. A.; Cribbs, L. L.; Koob, M. D.; Piedras-Renteria, E. S., American Journal of Physiology - Cell Physiology
  • Kelch-like 1 protein upregulates T-type currents by an actin-F dependent increase in alpha(1H) channels via the recycling endosome Aromolaran, K. A.; Benzow, K. A.; Cribbs, L. L.; Koob, M. D.; Piedras-Renteria, E. S., Channels (Austin, Tex.)
  • Ca2+ sparks and cellular distribution of ryanodine receptors in developing cardiomyocytes from rat. Snopko, R. M.; Ramos-Franco, J.; Di Maio, A.; Karko, K. L.; Manley, C.; Piedras-Renteria, E. S.; Mejia-Alvarez, R., Journal of Molecular & Cellular Cardiology
  • Altered frequency-dependent inactivation and steady-state inactivation of polyglutamine-expanded alpha1A in SCA6. Chen, H.; Piedras-Renteria, E. S., American Journal of Physiology - Cell Physiology
  • The Kelch-like protein 1 modulates P/Q-type calcium current density. Aromolaran, K. A.; Benzow, K. A.; Koob, M. D.; Piedras-Renteria, E. S., Neuroscience
  • 15th Annual Spring Brain Conference March 10-13, 2004, summary report., Anstrom, K.; Bergles, D.; Bradley, K.; Cao, Y.; Cestnick, L.; Lee, S.; Piedras-Renteria, E. S.; Savoie, K.; Smith, S. M., Somatosensory & motor research
  • Presynaptic Ca2+ channels compete for channel type-preferring slots in altered neurotransmission arising from Ca2+ channelopathy. Cao, Y. Q.; Piedras-Renteria, E. S.; Smith, G. B.; Chen, G.; Harata, N. C.; Tsien, R. W., Neuron
  • Presynaptic homeostasis at CNS nerve terminals compensates for lack of a key Ca2+ entry pathway. Piedras-Renteria, E. S.; Pyle, J. L.; Diehn, M.; Glickfeld, L. L.; Harata, N. C.; Cao, Y.; Kavalali, E. T.; Brown, P. O.; Tsien, R. W., Proceedings of the National Academy of Sciences of the United States of America
  • Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels. Urbano, F. J.; Piedras-Renteria, E. S.; Jun, K.; Shin, H. S.; Uchitel, O. D.; Tsien, R. W., Proceedings of the National Academy of Sciences of the United States of America
  • alpha- and betaCaMKII. Inverse regulation by neuronal activity and opposing effects on synaptic strength. Thiagarajan, T. C.; Piedras-Renteria, E. S.; Tsien, R. W., Neuron
  • Increased expression of alpha 1A Ca2+ channel currents arising from expanded trinucleotide repeats in spinocerebellar ataxia type 6. Piedras-Renteria, E. S.; Watase, K.; Harata, N.; Zhuchenko, O.; Zoghbi, H. Y.; Lee, C. C.; Tsien, R. W., Journal of Neuroscience