Richard Schultz
  • Professor
  • Microbiology and Immunology
Research Keywords
  • Cancer/Carcinogenesis
  • Proteins and Macromolecules

Heckathorn disease revisitedNand,S.; Barton,K.; Schultz,R.; Klein,M.; Kini,A.Blood 2014 ;123(9):1429-1431

Chromosome cohesion decreases in human eggs with advanced maternal ageDuncan,F. E.; Hornick,J. E.; Lampson,M. A.; Schultz,R. M.; Shea,L. D.; Woodruff,T. K.Aging Cell 2012 ;11(6):1121-1124

Coping with endometriosis.Kaatz,J.; Solari-Twadell,P. A.; Cameron,J.; Schultz,R.JOGNN - Journal of Obstetric, Gynecologic, & Neonatal Nursing 2010 ;39(2):220-225

Gene expression profiling of mouse oocytes and preimplantation embryosDuncan,F. E.; Schultz,R. M.Methods in enzymology 2010 ;477( ):457-480

Estrogen receptor inhibits c-Jun-dependent stress-induced cell death by binding and modifying c-Jun activity in human breast cancer cells.Qi,X.; Borowicz,S.; Pramanik,R.; Schultz,R. M.; Han,J.; Chen,G.Journal of Biological Chemistry 2004 ;279(8):6769-6777

p38 MAPK activation selectively induces cell death in K-ras-mutated human colon cancer cells through regulation of vitamin D receptor.Qi,X.; Tang,J.; Pramanik,R.; Schultz,R. M.; Shirasawa,S.; Sasazuki,T.; Han,J.; Chen,G.Journal of Biological Chemistry 2004 ;279(21):22138-22144

p38 isoforms have opposite effects on AP-1-dependent transcription through regulation of c-Jun. The determinant roles of the isoforms in the p38 MAPK signal specificity.Pramanik,R.; Qi,X.; Borowicz,S.; Choubey,D.; Schultz,R. M.; Han,J.; Chen,G.Journal of Biological Chemistry 2003 ;278(7):4831-4839

The p38 and JNK pathways cooperate to trans-activate vitamin D receptor via c-Jun/AP-1 and sensitize human breast cancer cells to vitamin D(3)-induced growth inhibitionQi,X.; Pramanik,R.; Wang,J.; Schultz,R. M.; Maitra,R. K.; Han,J.; DeLuca,H. F.; Chen,G.The Journal of biological chemistry 2002 ;277(29):25884-25892